Quantum Information Processing Model Explains Early and Recent Genome Repair Mechanisms
Molecular clocks exhibit time-dependent substitutions, ts, and deletions, td, as consequences of enzymatic processing of quantum informational content embodied within entangled proton qubit base pair super positions, G′-C′, *G-*C and *A-*T. These heteroduplex heterozygote point, r+/ rII, lesions are consequences of metastable hydrogen bonding amino (− NH2) genome protons encountering quantum uncertainty limits, Δx Δpx ≥ Ñ/2, which generate EPR arrangements, keto-amino â(entanglement)→ enol− imine, where reduced energy product protons are each shared between two indistinguishable sets of intramolecular electron lone-pairs belonging to enol oxygen and imine nitrogen on opposite strands, and thus, participate in entangled quantum oscillations at ~ 4×1013 s−1 (~ 4800 m s−1) between near symmetric energy wells in decoherence-free subspaces until “measured”, in a genome groove, δt<< 10−13 s, by a “truncated” Grover’s quantum bio-processor. Evidence demonstrates entangled proton qubit superpositions are transparent to “regular” DNA repair, but are detected and processed by an “earlier evolved” RNA repair system that implemented ancestral ribozyme – proton entanglement algorithms to introduce ts and td. These “repairs” of entangled superpositions allowed ancestral RNA genomes to avoid evolutionary extinction by disallowing duplication when ts + td exceeded a threshold limit. Natural selection introduced entanglement state bio-processor algorithms that provided a selective advantage for the duplex RNA genome. When duplex RNA became too “unwieldy”, rudimentary repair systems were introduced, which selected the more “suitable” DNA double helix over duplex RNA. Consequently, accumulated heteroduplex heterozygote superpositions are processed by “earlier evolved” enzyme-proton entanglement algorithms which introduce “new” ts or td, i.e., stochastic mutations.
Grant Cooper W